Tamia K. Lapointe
- Courses1
- Reviews1
- School: University of Calgary
- Campus:
- Department: Medicine
- Email address: Join to see
- Phone: Join to see
-
Location:
Calgary, AB - Dates at University of Calgary: April 2014 - April 2014
- Office Hours: Join to see
Biography
University of Calgary - Medicine
Lecturer/Course coordinator at Eberhard Karls Universität Tübingen, Germany
Research
Tamia K.
Lapointe
Stuttgart Area, Germany
I am an experienced lecturer and course coordinator with expertise in course design for both undergraduate and graduate modules. My area of expertise lies in human pathophysiology and immunology, but I also teach applied courses on advance scientific writing and literature analysis. I use a problem-based learning approach in most of my classes, giving the students the opportunity to go beyond the mindset of “read, memorize and repeat”, and focus on the development of their critical thinking abilities.
Experience
University of Calgary
Lecturer & Instructor
Graduate level courses:
- MDSC637 - Physiology of the Gastrointestinal Tract (Course coordinator)
- MDSC638 - Mucosal pathophysiology
Undergraduate level courses:
- MDSC203 - Developing Health Research Literacy I
- MDSC404 - Integrative Human Physiology
- ZOOL463 - Animal Physiology IIUniversity of Calgary
Teaching assistant
BIOL231 - Introduction to Cellular Biology
University of Calgary
Postdoctoral Research Fellow
Mechanisms of neurogenic inflammation, neuronal sensitization, and chronic pain in the contaxt of inflammatory bowel disease (Advisor: Dr. Christophe Altier)
Canadian Association of Gastroenterology
Committee Member for Research Affairs
Tamia worked at Canadian Association of Gastroenterology as a Committee Member for Research Affairs
University of Tübingen
Lecturer & Course coordinator
- Cellular and Molecular Basis of Inflammation (Graduate level course)
- Gastrointestinal Pathophysiology (Graduate level course)
- Introduction to Scientific Literature (Undergraduate level course)
- Advance Science Writing (Undergraduate level course)
Education
The University of Calgary
University Teaching Certificate
The University of Calgary
Doctor of Philosophy (PhD)
Gastrointestinal Pathophysiology
Thesis: The effect of Helicobacter pylori and proinflammatory mediators on gastrointestinal epithelia (Advisor: Dr. André G. Buret)Université du Québec à Montréal
Bachelor of Science (BSc)
Biology (Toxicology & Environmental Health)University of Calgary
Lecturer & Instructor
Graduate level courses: - MDSC637 - Physiology of the Gastrointestinal Tract (Course coordinator) - MDSC638 - Mucosal pathophysiology Undergraduate level courses: - MDSC203 - Developing Health Research Literacy I - MDSC404 - Integrative Human Physiology - ZOOL463 - Animal Physiology IIUniversity of Calgary
Teaching assistant
BIOL231 - Introduction to Cellular BiologyUniversity of Calgary
Postdoctoral Research Fellow
Mechanisms of neurogenic inflammation, neuronal sensitization, and chronic pain in the contaxt of inflammatory bowel disease (Advisor: Dr. Christophe Altier)
Publications
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Mechanisms by which inflammation may increase intestinal cancer risk in inflammatory bowel disease.
Inflammatory Bowel Disease
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Mechanisms by which inflammation may increase intestinal cancer risk in inflammatory bowel disease.
Inflammatory Bowel Disease
The role of TRPA1 in visceral inflammation and pain.
Channels
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Mechanisms by which inflammation may increase intestinal cancer risk in inflammatory bowel disease.
Inflammatory Bowel Disease
The role of TRPA1 in visceral inflammation and pain.
Channels
TRPM8 activation attenuates inflammatory responses in mouse models of colitis.
Proc Natl Acad Sci U S A.
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Mechanisms by which inflammation may increase intestinal cancer risk in inflammatory bowel disease.
Inflammatory Bowel Disease
The role of TRPA1 in visceral inflammation and pain.
Channels
TRPM8 activation attenuates inflammatory responses in mouse models of colitis.
Proc Natl Acad Sci U S A.
New neostigmine-based behavioral mouse model of abdominal pain
Pharmacological Reports
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Mechanisms by which inflammation may increase intestinal cancer risk in inflammatory bowel disease.
Inflammatory Bowel Disease
The role of TRPA1 in visceral inflammation and pain.
Channels
TRPM8 activation attenuates inflammatory responses in mouse models of colitis.
Proc Natl Acad Sci U S A.
New neostigmine-based behavioral mouse model of abdominal pain
Pharmacological Reports
Epithelial dysfunction in the pathogenesis of Helicobacter pylori infection (chapter 7).
In: Bacteriology Research Developments - Helicobacter Pylori: Detection Methods, Diseases and He Nova alth Implications. Manfredi M, de’Angelis, ed. New York:Biomedical. pp. 115-144
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Mechanisms by which inflammation may increase intestinal cancer risk in inflammatory bowel disease.
Inflammatory Bowel Disease
The role of TRPA1 in visceral inflammation and pain.
Channels
TRPM8 activation attenuates inflammatory responses in mouse models of colitis.
Proc Natl Acad Sci U S A.
New neostigmine-based behavioral mouse model of abdominal pain
Pharmacological Reports
Epithelial dysfunction in the pathogenesis of Helicobacter pylori infection (chapter 7).
In: Bacteriology Research Developments - Helicobacter Pylori: Detection Methods, Diseases and He Nova alth Implications. Manfredi M, de’Angelis, ed. New York:Biomedical. pp. 115-144
TRPV1 sensitization mediates post-inflammatory visceral pain following acute colitis.
Am J Physiol Gastrointest Liver Physiol
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Mechanisms by which inflammation may increase intestinal cancer risk in inflammatory bowel disease.
Inflammatory Bowel Disease
The role of TRPA1 in visceral inflammation and pain.
Channels
TRPM8 activation attenuates inflammatory responses in mouse models of colitis.
Proc Natl Acad Sci U S A.
New neostigmine-based behavioral mouse model of abdominal pain
Pharmacological Reports
Epithelial dysfunction in the pathogenesis of Helicobacter pylori infection (chapter 7).
In: Bacteriology Research Developments - Helicobacter Pylori: Detection Methods, Diseases and He Nova alth Implications. Manfredi M, de’Angelis, ed. New York:Biomedical. pp. 115-144
TRPV1 sensitization mediates post-inflammatory visceral pain following acute colitis.
Am J Physiol Gastrointest Liver Physiol
Helicobacter pylori activates calpain via toll-like receptor 2 to disrupt adherens junctions in human gastric epithelial cells.
Infection and Immunity
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Mechanisms by which inflammation may increase intestinal cancer risk in inflammatory bowel disease.
Inflammatory Bowel Disease
The role of TRPA1 in visceral inflammation and pain.
Channels
TRPM8 activation attenuates inflammatory responses in mouse models of colitis.
Proc Natl Acad Sci U S A.
New neostigmine-based behavioral mouse model of abdominal pain
Pharmacological Reports
Epithelial dysfunction in the pathogenesis of Helicobacter pylori infection (chapter 7).
In: Bacteriology Research Developments - Helicobacter Pylori: Detection Methods, Diseases and He Nova alth Implications. Manfredi M, de’Angelis, ed. New York:Biomedical. pp. 115-144
TRPV1 sensitization mediates post-inflammatory visceral pain following acute colitis.
Am J Physiol Gastrointest Liver Physiol
Helicobacter pylori activates calpain via toll-like receptor 2 to disrupt adherens junctions in human gastric epithelial cells.
Infection and Immunity
Interleukin-18 facilitates neutrophil transmigration via myosin light chain kinase-dependent disruption of occludin, without altering epithelial permeability
Am J Physiol Gastrointest Liver Physiol.
Sustained neurochemical plasticity in central terminals of mouse DRG neurons following colitis
Cell & Tissue Research
Mechanisms by which inflammation may increase intestinal cancer risk in inflammatory bowel disease.
Inflammatory Bowel Disease
The role of TRPA1 in visceral inflammation and pain.
Channels
TRPM8 activation attenuates inflammatory responses in mouse models of colitis.
Proc Natl Acad Sci U S A.
New neostigmine-based behavioral mouse model of abdominal pain
Pharmacological Reports
Epithelial dysfunction in the pathogenesis of Helicobacter pylori infection (chapter 7).
In: Bacteriology Research Developments - Helicobacter Pylori: Detection Methods, Diseases and He Nova alth Implications. Manfredi M, de’Angelis, ed. New York:Biomedical. pp. 115-144
TRPV1 sensitization mediates post-inflammatory visceral pain following acute colitis.
Am J Physiol Gastrointest Liver Physiol
Helicobacter pylori activates calpain via toll-like receptor 2 to disrupt adherens junctions in human gastric epithelial cells.
Infection and Immunity
Interleukin-18 facilitates neutrophil transmigration via myosin light chain kinase-dependent disruption of occludin, without altering epithelial permeability
Am J Physiol Gastrointest Liver Physiol.
Granulocyte-colony-stimulating factor (G-CSF) signaling in spinal microglia drives visceral sensitization following colitis.
Proc Natl Acad Sci U S A.
