Resume

• 2004

  Ph.D.

  Behavioral Neuroscience

  The University of Connecticut

• Behavioral Neuroscience

  Research

  Statistics

  University Teaching

  Data Analysis

  Pharmacology

  Science

  SPSS

  Neuroscience

  Psychology

  Pharmaceutical Industry

  Life Sciences

  Higher education

  Teaching

  Animal Models

  HPLC

  Scientific Writing

  High-Performance Liquid Chromatography (HPLC)

  behavioral pharmacology

  Statistical Data Analysis

  Nucleus accumbens and effort-related functions: behavioral and neural markers of the interactions between adenosine A2A and dopamine D2 receptors.

  Nucleus accumbens and effort-related functions: behavioral and neural markers of the interactions between adenosine A2A and dopamine D2 receptors.

  Cognitive Training at a Young Age Attenuates Deficits in the zQ175 Mouse Model of HD.

  Deficits in a Simple Visual Go/No-go Discrimination Task in Two Mouse Models of Huntington's Disease.

  Reduced striatal acetylcholine efflux in the R6/2 mouse model of Huntington's disease: an examination of the role of altered inhibitory and excitatory mechanisms.

  Cognitive deficits in transgenic and knock-in HTT mice parallel those in Huntington's disease.

  Intra-accumbens injections of the adenosine A2A agonist CGS 21680 affect effort-related choice behavior in rats.

  In-vitro analysis of Pitx3 in mesodiencephalic dopaminergic neuron maturation.

  Effect of the adenosine A2A receptor antagonist MSX-3 on motivational disruptions of maternal behavior induced by dopamine antagonism in the early postpartum rat.

  Dopamine/adenosine interactions related to locomotion and tremor in animal models: possible relevance to parkinsonism.

  Genetic deletion of transglutaminase 2 does not rescue the phenotypic deficits observed in R6/2 and zQ175 mouse models of Huntington's disease.

  The adenosine A2A antagonist MSX-3 reverses the effort-related effects of dopamine blockade: differential interaction with D1 and D2 family antagonists.

  Stimulant effects of adenosine antagonists on operant behavior: differential actions of selective A2A and A1 antagonists.

  Nucleus accumbens adenosine A2A receptors regulate exertion of effort by acting on the ventral striatopallidal pathway.

  Differential actions of adenosine A1 and A2A antagonists on the effort-related effects of dopamine D2 antagonism.

  Systemic administration of the adenosine A(2A) agonist CGS 21680 induces sedation at doses that suppress lever pressing and food intake.

  Temporal Control Deficits in Murine Models of Huntington's Disease

  Cognitive Deficits in the R6/2 mouse model of Huntington’s disease and their Amelioration with Donepezil.

  Dopamine

  behavioral economics

  and effort.

  Role of dopamine-adenosine interactions in the brain circuitry regulating effort-related decision making: insights into pathological aspects of motivation

  Forebrain circuitry involved in effort-related choice: Injections of the GABAA agonist muscimol into ventral pallidum alter response allocation in food-seeking behavior.

  Andrew

  Farrar

  University of Connecticut

  University of Massachusetts Amherst

  Rutgers University - Newark

  PsychoGenics

  Newark

  NJ

  Postdoctoral Associate

  Rutgers University - Newark

  Amherst

  Massachusetts

  •Developed and taught intermediate and advanced courses in Neuroscience

  serving students from diverse majors

  including Psychology

  Biology

  Biochemistry and Kinesiology. \n•Courses include Drugs and Behavior

  Behavioral Neuroendocrinology

  Lab in Neuroscience and Junior Writing.

  Lecturer

  University of Massachusetts Amherst

  PhD in Behavioral Neuroscience

  advisor: Dr. John D. Salamone.

  University of Connecticut

  Research Scientist

  •Developed and delivered research proposals

  results and final study reports in client-facing role.\n•Worked with KOLs in industry

  non-profit and academic sectors to establish research priorities and develop strategic plans.\n•Led team of research associates to execute complex studies for diverse clients in biotechnology and pharmaceutical sectors; collaborated with internal and external partners to deliver high quality results.\n•Developed

  validated and implemented experimental protocols for phenotype assessment of mouse models of neurological and psychiatric disease; streamlined existing platforms to increase throughput and efficiency.

  PsychoGenics

  Consultant

  \n

  PsychoGenics

  Research Fellow

  • Participated in design

  execution and analysis of behavioral neuroscience experiements in new faculty laboratory. \n•Served as neuroscience representative to Models to Medicine Center within the UMass Institute for Applied Life Sciences (IALS); collaborated with internal stakeholders to draft strategic planning documents and presented institutional neuroscience research capabilities to external Industry Advisory Board.\n• Trained undergraduate and graduate students in laboratory procedures.\n

  University of Massachusetts Amherst